Fascinating Aspects of Glycobiology—Part 7 (COVID-19)
Author - Larry A. Law
July 22, 2025
Larry A. Law
| Saying the word COVID-19
still gets my blog articles tagged by social media and sent to spam folders when I include them in the eZine. It's been five years, but the entrenched medical system still does not want the truths to come out concerning the fake pandemic. I have published 125 articles since November 2019 when the medically-induced fearmongering began. Fortunately, a positive side-effect of the panic resulted in the public learning more about glycobiology
than they had ever known before.
|
Glycobiology and COVID-19
As shown in the picture above, some of the areas specifically related to glycobiology and COVID-19 included: the SARS-CoV-2
(a glycoprotein), cell receptors (
—also a glycoprotein), the experimental
which forced cells to create a synthetic spike glycoprotein, how chronic disease
, and the
.
Dr. Fauci Needs to be Held Accountable
The Telegraph,
"COVID vaccines may have helped fuel rise in excess deaths."
A great new movie documenting issues surrounding the COVID pandemic is called
Unsafe and Ineffective
. You can watch it for free by
and putting in your name and email.
Hydroxychloroquine Example of Glycobiology
Zinc Ionophore
HCQ) acts as a zinc
ionophore
(carrier) on the glycocalyx of the cells. Zinc is a crucial mineral in fighting viruses. It interrupts the ability of the virus to replicate. The zinc ion has 2 positive charges (red dot in picture below). The cell
membrane
has no charge, so that the zinc ion cannot cross through that barrier by itself. HCQ is used by the cell and becomes embedded within the cell membrane (glycocalyx) as an ionophore (green revolving door in picture below). This carrier can capture a zinc ion floating outside the cell. The carrier then encapsulates the zinc ion, allowing it to keep its charge. It transfers the zinc from outside the cell to the inside, where it is released. The positively charged zinc ion can now interact with the virus's RdRp enzyme, independent of HCQ, and alter it so it cannot do its job. In this way, replication of the virus ceases because of the disruption of this important viral metabolic pathway. The virus cannot reproduce inside the cell.
Glycosylation
ACE2
glycocalyx
spike glycoprotein
affinity of binding
slips
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